StackTerminal.Health

Boron

Boron is a trace element that modulates sex hormone binding globulin, increases free testosterone and estradiol, enhances vitamin D activity, and supports calcium and magnesium retention in bone; evidence suggests benefits for hormonal balance and bone mineral metabolism.

hormonalbonetestosteronemetabolicmineral-metabolism
Dosing model
FLATFixed dose (no body-weight scaling).
Min dose
3 mg
Max dose
10 mg
Rounding
1 mg
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Evidence
2 records
Free testosterone, estradiol, and inflammatory cytokines in healthy men
Healthy male volunteers • controlled supplementation study
Low

A controlled study in 8 healthy men found that daily supplementation with 10 mg boron for one week significantly increased free testosterone (11.83 to 15.18 pg/mL), decreased estradiol (42.33 to 25.81 pg/mL), increased dihydrotestosterone, cortisol, and vitamin D, and decreased all three measured inflammatory biomarkers (hsCRP, TNF-α, IL-6).

Dose: 10 mg • Duration: 1 week
Citation: Naghii MR, Mofid M, Asgari AR, Hedayati M, Daneshpour MS. Comparative effects of daily and weekly boron supplementation on plasma steroid hormones and proinflammatory cytokines. J Trace Elem Med Biol. 2011;25(1):54-8.
Small pilot study (n=8); findings are preliminary. Larger RCTs needed.
Calcium and bone mineral metabolism in postmenopausal women
Postmenopausal women • controlled metabolic study
Low

A controlled metabolic study found that boron supplementation (3 mg/day) in postmenopausal women markedly reduced urinary calcium and magnesium excretion and significantly elevated serum 17β-estradiol and testosterone; effects were most pronounced in women consuming low-magnesium diets, suggesting boron potentiates calcium retention and bone-protective hormonal profiles.

Dose: 3 mg • Duration: 4 weeks
Citation: Nielsen FH, Hunt CD, Mullen LM, Hunt JR. Effect of dietary boron on mineral, estrogen, and testosterone metabolism in postmenopausal women. FASEB J. 1987;1(5):394-7.
Foundational metabolic unit study; supports boron's role in bone health via hormonal and mineral mechanisms.
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